Moreover, if the FALC frequent is raised, associating it high indices of the GGT/FA, these is more suggestive of DHA of what not alcoholic heptica illness (Mincis, 2010). It is important to detach that, due to discrepancy between clinical manifestations and alterations biochemists and the degree of histopatolgica injury, the monitorizao of the alcoholic heptica illness is basically morphologic, having the heptica bipsia formal indication (Son, 2006). However, for Mincis (2010), the morphologic data, without the knowledge of the clinical data (especially the etlicos habits) do not make possible the diagnosis of ‘ ‘ heptica illness of etiology alcolica’ ‘. Credit: Mayo Clinic-2011. Moreover, the data of the histolgico examination do not inform on the heptica disfuno (and in this manner they do not substitute the called tests of heptica function). Still in relation to the diagnosis, it is standed out use of tumorais markers (or biological markers), that they are macro-molecules gifts in the tumor, the blood or other biological liquids, whose appearance and or alterations in its concentrations is related with gnese and the growth of neoplsicas cells (Capezzoli, 2001). The therapeutical boarding of the related pathology involves clinical and/or surgical interventions. (Not to be confused with endocrinologist!). According to Mincis (2010), not specific the clinical treatment is based on the total drink abstention alcoholic with significant regression of esteatose and increase of the supervened one between the patients who present the compensated form of the illness. However, it is important to stand out, the occurrence in some patients of agitation, anxiety, alterations of mood (irritability, disforia), tremors, nauseas, vomits, taquicardia, arterial hipertenso, hallucinations, Delirium Tremens (DT), convulsions, configuring itself as syndrome of the alcoholic abstinence (SAA), requiring the use of benzodiazepnicos (et.al Laranjeira, 2000).. .